Commentaries on point: Counterpont: Exercise training-induced bradycardia

Leicht, Anthony S., Boullosa, Daniel A., and Hautala, Arto J. (2017) Commentaries on point: Counterpont: Exercise training-induced bradycardia. Journal of Applied Physiology, 123 (3). p. 693.

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[Extract] TO THE EDITOR: In the recent Point:Counterpoint debate (2, 3), both research groups argued that training-induced bradycardia was a result of changes in intrinsic, sinoatrial node firing (i.e., intrinsic rate) or cardiac autonomic/parasympathetic regulation. While Billman (2) stated the possibility of a combination of these mechanisms, no further discussion of this was provided by either research group. Rather, the discussion focused on either mechanism and we would urge researchers to consider a more complex scenario – contribution of either mechanism that is moderated by other factors, or both mechanisms, potentially in combination with other elements (e.g., cardiac tructural changes). For example, training-induced bradycardia was reported in young adults with no changes in heart rate variability (HRV)(4), supporting an intrinsic rate mechanism. However, bradycardia was induced similarly with enhanced HRV in young adults following high-intensity exercise, supporting a cardiac autonomic mechanism (5).

Item ID: 52124
Item Type: Article (Commentary)
ISSN: 1522-1601
Date Deposited: 23 Jan 2018 03:37
FoR Codes: 11 MEDICAL AND HEALTH SCIENCES > 1106 Human Movement and Sports Science > 110602 Exercise Physiology @ 100%
SEO Codes: 97 EXPANDING KNOWLEDGE > 970106 Expanding Knowledge in the Biological Sciences @ 100%
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